Serum Antibodies against Human Intracisternal A-type Particle (HIAP) Endogenous Retrovirus in Alopecia Areata Patients: A Hallmark of Autoimmune Disease?
نویسندگان
چکیده
منابع مشابه
Serum antibody reactivity to human intracisternal A-type particle retrovirus proteins in systemic sclerosis patients.
Serum antibodies against human intracisternal A-type particle (HIAP) endogenous retrovirus have been found to be associated with various autoimmune pathologies. To evaluate the presence of serum antibody reactivity to HIAP proteins in systemic sclerosis, a Western blot analysis was performed on sera from 42 patients with systemic sclerosis, in comparison with 18 sera from patients with primary ...
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Background: Autoimmune mechanisms are involved in the etiology of Alopecia areata. It is also shown that Selenium has some effects on regulation of autoimmune mechanisms. Objective: To compare the plasma level of Selenium between those with and without Alopecia areata. Patients and Methods: In this case-control study, 29 patients with Alopecia areata referred to Razi hospital in Tehran, a...
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Introduction: Alopecia Areata is a chronic recurrent nonscarring skin disease characterized by sudden Asymptomatic localized hairless. Alopecia Areata can cause emotional and psychosocial distress in affected individuals. Personal appearance due to patchy hairloss may affect patients social relationship and change in quality of life. This study is designed to investigate the effect of Ale...
متن کاملThe autoimmune basis of alopecia areata: a comprehensive review.
Alopecia areata (AA) is a common, non-scarring dermatologic condition regularly distinguished by patches of hair loss on the scalp also manifesting in other, severe forms, including alopecia totalis (total loss of hair on the scalp) and alopecia universalis (complete loss of hair on the scalp and body). AA is a clinically heterogeneous disease with greatly varying yet typical symptoms, but the ...
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ژورنال
عنوان ژورنال: Journal of Investigative Dermatology
سال: 2004
ISSN: 0022-202X
DOI: 10.1111/j.0022-202x.2004.23216.x